The respiratory system allows the entry of O 2 and the parting of CO 2 in the body. The proper function of tissues and organs requires adequate oxygen from the lungs into the blood. Oxygen utilization and carbon dioxide production are, therefore, essential to life. Ideally, in humans and all mammals, oxygen (O 2) from the air is breathed into the lungs, while carbon dioxide (CO 2) is breathed out as a waste product made by cells of the body. The chapter will, therefore, provide more insights into the pathophysiology, clinical attributes, pathogenesis, and treatment approach of respiratory failure in COVID-19 conditions, as well as evaluate any similarities and differences that may exist.īreathing is an indispensable requirement in life. Knowing the exact phenotype in patients with COVID-19 has been of important clinical significance in seeking the right treatment strategies for treating respiratory failure. These features are heterogeneous and categorized into progressive respiratory distress and unique “silent hypoxemia” as two phenotypes. Disorders of the respiratory neuromuscular, airway, pulmonary vesicles, and lung parenchyma all manifest in COVID-19. Respiratory failure instigated by COVID-19 has some clinical features in affected patients. Simply put, respiratory failure is the major clinical demonstration of COVID-19 and the frontline cause of the associated mortality. The coronavirus disease-2019 (COVID-19) is majorly a respiratory disease capable of causing infection caused by the newly discovered coronavirus (SARS-CoV-2) with a consequential effect on respiratory failure. Respiratory failure, characterized as the unsuccessful maintenance of adequate gas exchange, is associated with abnormalities of arterial blood gas tensions.
0 Comments
Leave a Reply. |
AuthorWrite something about yourself. No need to be fancy, just an overview. ArchivesCategories |